Research: Differential Recognition of Mtb-specific Epitopes as a Function of Tuberculosis Disease History

07 Aug 2017
Differential Recognition of Mycobacterium tuberculosis-Specific Epitopes as a Function of Tuberculosis Disease History
07 Aug 2017

SATVI authors Professor Mark Hatherill, Associate Professor Tom Scriba,  Drs. Munyaradzi Musvosvi and Virginie Rozot have co-authored a research paper titled "Differential recognition of Mtb-specific epitopes as a function of tuberculosis disease history" appearing in the American Journal of Respiratory and Critical Care Medicine.

Differential recognition of Mycobacterium tuberculosis (Mtb) epitopes correlates with conservation in the microbiome and less so in nontuberculous mycobacteria (NTM).
Photo Matt Feldman, AERAS.

 

Abstract

Rationale: Individuals with a history of tuberculosis (TB) disease are at elevated risk of disease recurrence. The underlying cause is not known, but one explanation is that previous disease results in less-effective immunity against Mycobacterium tuberculosis (Mtb).

 

We hypothesized that the repertoire of Mtb-derived epitopes recognized by T cells from individuals with latent Mtb infection differs as a function of previous diagnosis of active TB disease.

Objectives: We hypothesized that the repertoire of Mtb-derived epitopes recognized by T cells from individuals with latent Mtb infection differs as a function of previous diagnosis of active TB disease.

Methods: T-cell responses to peptide pools in samples collected from an adult screening and an adolescent validation cohort were measured by IFN-γ enzyme-linked immunospot assay or intracellular cytokine staining.

Measurements and main results: We identified a set of "type 2" T-cell epitopes that were recognized at 10-fold-lower levels in Mtb-infected individuals with a history of TB disease less than 6 years ago than in those without previous TB. By contrast, "type 1" epitopes were recognized equally well in individuals with or without previous TB. The differential epitope recognition was not due to differences in HLA class II binding, memory phenotypes, or gene expression in the responding T cells. Instead, "TB disease history-sensitive" type 2 epitopes were significantly (P < 0.0001) more homologous to sequences from bacteria found in the human microbiome than type 1 epitopes.

Differential recognition of Mycobacterium tuberculosis (Mtb) epitopes correlates with conservation in the microbiome and less so in nontuberculous mycobacteria (NTM).
Figure. Differential recognition of Mycobacterium tuberculosis (Mtb) epitopes correlates with conservation in the microbiome and less so in nontuberculous mycobacteria (NTM).
Conservation of type 1 (n = 113; white dots) and type 2 (n = 123; gray dots) epitopes in (A) the human microbiome, (B) Mtb complex strains, and (C) NTM strains is shown. Each dot represents the maximum BLOSUM score per individual peptide (i.e., the highest homology found for each peptide within the human microbiome, Mtb complex strains, or NTM strains). Median and interquartile range are indicated. The numbers above the brackets are P values determined by two-tailed Mann-Whitney test.
Conclusions: Preferential loss of T-cell reactivity to Mtb epitopes that are homologous to bacteria in the microbiome in persons with previous TB disease may reflect long-term effects of antibiotic TB treatment on the microbiome.

 

Keywords: Mycobacterium tuberculosis; T-cell epitopes; adaptive immunity; microbiome; tuberculosis.

 

Citation: Scriba TJ, Carpenter C, Pro SC, Sidney J, Musvosvi M, Rozot V, Seumois G, Rosales SL, Vijayanand P, Goletti D, Makgotlho E, Hanekom W, Hatherill M, Peters B, Sette A, Arlehamn CSL. 2017. Differential Recognition of Mycobacterium tuberculosis-Specific Epitopes as a Function of Tuberculosis Disease History. American Journal of Respiratory Crit Care Med. 2017 Sep 15;196(6):772-781. Click here.